he coloration patterns in Siamese Cats arise from a temperature-sensitive mutation. An enzyme required for the synthesis of dark pigment in the fur is unable to function in areas close to the body where normal physiological temperatures prevail. However, at the tips of the ears, paws, the tip of the tail and other extremities where the temperature is slightly lower, the enzyme works correctly and dark pigment is produced. What is happening with the enzyme at the molecular level that explains this? What kind of interactions might be altered by the mutation?

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